Ursodeoxycholic acid protects cardiomyocytes against cobalt chloride induced hypoxia by regulating transcriptional mediator of cells stress hypoxia inducible factor 1α and p53 protein

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Ursodeoxycholic acid protects cardiomyocytes against cobalt chloride induced hypoxia by regulating transcriptional mediator of cells stress hypoxia inducible factor 1α and p53 protein

In hepatocytes, ursodeoxycholic acid (UDCA) activates cell signalling pathways such as p53, intracellular calcium ([Ca2+]i), and sphingosine-1-phosphate (S1P)-receptor via Gαi-coupled-receptor. Recently, UDCA has been shown to protect the heart against hypoxia-reoxygenation injury. However, it is no...

وصف كامل

التفاصيل البيبلوغرافية
الحاوية / القاعدة:	Cell Biochemistry and Function
المؤلف الرئيسي:	Mohamed A.S.; Hanafi N.I.; Sheikh Abdul Kadir S.H.; Md Noor J.; Abdul Hamid Hasani N.; Ab Rahim S.; Siran R.
التنسيق:	مقال
اللغة:	English
منشور في:	John Wiley and Sons Ltd 2017
الوصول للمادة أونلاين:	https://www.scopus.com/inward/record.uri?eid=2-s2.0-85031325768&doi=10.1002%2fcbf.3303&partnerID=40&md5=76edac7ed2f6e599351797d27545b12b

مواد مشابهة

Ursodeoxycholic acid regulates caspase-9 and ROS production in protecting cardiomyocytes against hypoxia
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Ursodeoxycholic acid upregulates ERK and Akt in the protection of cardiomyocytes against CoCl2
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